Acid Reflux, Indigestion & Food Stagnation
When the stomach is overwhelmed, indigestion ensues and food stagnates. This is why the most common type of acid reflux arises from heavy Kapha provoking foods such as cheeseburgers, burritos,
ice cream and fried foods. Food stagnates because digestive organs are programmed to hold back food until properly digested. Normally, when one stage of digestion is complete, peristalsis (regular wavelike muscular contractions of the intestines) propels the food further down the digestive tract. When indigestion delays food movement, bacteria feed on the partially undigested food and multiply.
Food becomes rotten, foul smelling and gaseous as it lays stagnant in the intestine. Stagnation in the stomach, a condition known as gastroparesis, causes a subjective feeling of heaviness, burping, upper GI (short for gastro-intestinal) distension and regurgitation of food mixed with acid. Rotten food also contains chemicals released by bacteria that may increase inflammation and degradation of tissue (gastritis). Indigestion may involve one or more doshas.
Kapha type indigestion is characterized by low metabolism and mucus obstruction. Low metabolism, perhaps due to hypothyroid, makes the digestive tract cold and sluggish, and contractions of stomach muscles weak. Mucus buildup in the stomach from a diet high in sweets, dairy, wheat, red meat, and refined sugars directly obstructs the action of digestive acids. Common symptoms of Kapha type acid reflux include a subjective feeling of heaviness, acid reflux that appears after late-night meals, morning mucus and congestion in the respiratory tract, as well as a thick coating on the tongue and a tendency to gain weight.
Gastritis
Gastritis is inflammation of the digestive tract. Gastritis ultimately leads to impairment of digestive function, a
Vata disorder. Infection, auto-immune disorders, loss of the protective mucus barrier, bile reflux and other pathological agents can cause gastritis. When inflamed, the folds in the stomach become enlarged and swollen. Common signs of gastritis include abdominal tenderness and teeth marks on the tongue border. One type of gastritis that causes acid reflux is chronic atrophic gastritis. Atrophic refers to 'atrophy, a medical term meaning loss of function. In atrophic gastritis, there is a loss of acid secreting cells of the stomach. The inability to secrete a normal quantity of acids is a condition known as hypochlorhydria or hypoacidity.
Infection with H. pylori, a gram negative bacteria, is a common cause of acute gastritis which may become chronic and atrophic. The organism is predominantly found beneath the gastric mucosal layer that lines the surface epithelium of the stomach. H. pylori produces abundant quantities of urease, an enzyme that functions to produce ammonia in order to neutralize gastric acid. Urease also inflames the gut wall. An interesting side note: although gastritis secondary to H. pylori infection may cause acid reflux, the neutralization of stomach acids by urease actually reduces symptoms of GERD including esophageal irritation.
The stomach is normally well protected by a thick layer of mucus. Dehydration may leave stomach glands unable to produce enough mucus. Alcohol, smoking, and spices, carbonic acid (in carbonated beverages) and hyperacidity can also erode the protective mucus barrier of the stomach, and then irritate the tissues beneath causing gastritis.
Digestion and the Blood
Most of the energy and fluids used for digestion are supplied through the blood. Thus, digestion is only as strong as the
blood and circulation. A high-
Vata lifestyle including overexertion and skipping meals deplete the blood. Stress effectively shuts down circulation to digestive organs. Cold temperature also constricts blood vessels and circulation. Anemia and other blood deficiencies can also cause cold temperature related circulation problems. Deficiency-related cold is associated with feeling cold often even during the summer, voiding a larger quantity of soft but formed stools, and clear, thin nasal discharge.
Dehydration reduces the production of digestive juices by the stomach, liver and pancreatic glands creating a condition of "Dry Stomach Syndrome". Consider that 2 liters of acid production a day could easily drain the blood of much needed fluids, especially in thin Vata individuals. Here are some signs of a dry stomach:
- If the tongue is dry, chances are the stomach is too.
- Dry skin is an indicator that all glands are dry, including stomach, liver, and pancreas.
- A small appetite could indicate insufficient acid production in the stomach.
- Bloating, gas and burping signify food remains partially undigested
Hypoacidity
The blood, circulatory and
gastritis based deficiencies discussed above reduce acid production, a condition called hypoacidity. Hypoacidity generally causes
Vata-type indigestion, characterized by frequent hunger but a feeling of fullness or heaviness after a small meal, which may be followed by upper abdominal distention, bloating and
burping. Hypoacidity related indigestion is a common, if not the leading, cause of acid reflux and should be ruled out before taking antacids.
Hyperacidity
Although most western medical doctors assume hyperacidity in all cases of acid reflux, we believe hyperacidity may be the exception, not the rule, in cases of acid reflux. Hyperacidity is fundamentally associated with aggravated Pitta. It results when the blood (
bhrajaka pitta) and the stomach (
pachaka pitta) are overactive, making a person 'hot blooded' both emotionally and physically. Typically, symptoms appear 1-2 hrs after eating. Vasodilation, thinning of the blood and increased cardiac output together increase the rate of digestion and can cause hyperacidity. Smoking,
alcohol, and excess use of
sour and spicy foods are just some of the ways to over-stimulate digestive juices.
Stress, worry, anger and frustration also increase production of stomach acids (Vata pushing Pitta). Emotional stress stimulates the vagus nerve, in turn stimulating secretion of gastrin and production of stomach acids. Gastrin also relaxes the lower esophageal sphincter (LES), leaving the esophagus vulnerable to acid reflux. Via the sympathetic nervous system, stress causes blood vessels to constrict, decreasing production and quality of mucus. In addition to directly causing acid reflux, more acid and less mucus leaves the stomach vulnerable to "stress gastritis".
Whereas heaviness and low appetite are commonly associated with hypoacidity, a person with chronic hyperacidity would likely have a large appetite and feel hungry between meals. The tongue will generally have a red tip or show other heat signs including a rapid, strong and bounding pulse. Hyperacidity will show fewer signs of indigestion-related gas and bloating.
Acid Reflux and Bile Reflux
Acid reflux must be differentiated from bile reflux. Bile reflux has similar symptoms to acid reflux. Bile is an alkaline salt produced by the
liver and is normally released by the gallbladder into the
small intestine. Then bile emulsifies fats for absorption into the lymphatic system. Sometimes, bile backs up through the pyloric valve into the stomach causing atrophic gastritis eventually leading to acid reflux.
Bile reflux may result from bile insufficiency (Vata in
ranjaka pitta),
gallbladder stasis (Kapha blocking
ranjaka pitta), or a defective pyloric valve, and any kind of food stagnation in the duodenum. The duodenum is the section of the
small intestine just after the stomach. Food stagnation in the duodenum signals the stomach to stop releasing food, causing backup and acid reflux.
Insufficient bile impairs digestion of fatty foods causing stagnation. If related to Vata deficiency, symptoms will include yellow, light colored stools, emotional instability, and hypogylcemia. Common signs of Pitta related bile obstruction are yellowing of the eyes, nails, or skin, secondary to liver distress and inflammation. Gall bladder stasis from Kapha occurs when thick bile gets stuck in the gallbladder leading to indigestion, irritability, red puffy cheeks or hands, and tiredness after eating fatty foods such as french fries or potato chips.
Western Medicine
Antacids reduce symptoms and irritation from acute acid regurgitation but they do not address the root cause. Antacids are often prescribed before determining whether the underlying cause is hyper or hypo-acidity. Antacids actually increase food stagnation and exacerbate chronic acid reflux in hypo-acidic clients. The following western pharmaceuticals can increase the risk of acid reflux:
Other causes of Acid Reflux
Hiatal hernia (a condition where a portion of the stomach protrudes upward into the chest), injury to the lower esophageal sphincter (LES), inflammation, and overeating can cause stomach contents to leak into the esophagus. Full or partial intestinal obstruction by compacted or otherwise stagnant fecal matter can cause backup. Increased pressure on abdominal organs during pregnancy can cause late term acid reflux. Strenuous exercise such as weight lifting increase the risk of acid reflux.
Hiccup, retching, spasms, and
vomiting cause the reverse flow and regurgitation of stomach contents.